Maternal-Fetal Medicine Doctors | NYU Langone Health
View all NYU Langone doctors who specialize in maternal-fetal medicine.
Maternal–Fetal Medicine Services | NYU Langone Health
Maternal–fetal medicine specialists at NYU Langone provide screening, diagnostic testing, and treatment during high-risk pregnancies.
Mechanisms and Enhancement of Learning During Sleep
There are two major steps in learning: 1) attending to and encoding the information and 2) stabilizing or consolidating the transiently encoded information. Memory consolidation, the second stage in the learning process, is the transformation of short-term memory traces into stable long-term representations. Sleep is increasingly recognized for its critical role in consolidation, perhaps by replaying, processing, and integrating temporary memories into long-term storage. In this study, we seek to elucidate how sleep supports learning across various cognitive domains, by relating aspects of sleep architecture (eg time spent in NREM vs REM sleep, spindle frequency) and fMRI connectivity to learning outcomes.
Mechanisms of Peripheral Vascular Disease
The proposed study will enable the use of human tissue samples and outcome data by basic and clinical investigators for research into the biology, causes, prevention and therapy of patients with documented peripheral arterial vascular disease secondary to atherosclerosis. The collection of paired information will provide important clinically relevant findings that will add to the growing understanding of the natural history of plaque formation in the arteries of the periphery and will enable to decipher the molecular mechanisms in this pathology. We will prospectively collect both tissue to study the mechanism of the natural history of PAD. We will also collect relevant clinical and medical information on these subjects and continue to collect follow-up information on the surgical patients using their medical records in order to follow their outcome.
Mechanisms of rAcial dIfferences in the relatioNship between Obstructive Sleep Apnea and in vivo Tau deposition in the context of AmYloid burden
African-Americans (blacks) have two times the risk of developing Alzheimer’s disease (AD) compared to non-Hispanic whites.1-6 Neuropathological studies show blacks with more mixed pathology1-6. Recent evidence demonstrate differences in AD biomarkers with blacks having decreased cortical thickness, and lower cerebrospinal fluid (CSF) P-tau, and T-tau.7-10. Notably, area-based socio-economic status (SES) partly explain racial differences in cortical thickness.11 This suggests the possible existence of additional physiologic differences on AD-risk by race, mediated by SES and resulting to greater neuronal loss, similar or less CSF-tau for similar levels of amyloid. Recent studies suggest that obstructive sleep apnea (OSA) increases AD-risk,12-14 is associated with higher brain amyloid and tau in cognitive normal (CN) participants.15-22. Notably, blacks have a higher burden of symptomatic OSA, particularly with excessive daytime sleepiness (EDS),23 which is associated with longitudinal amyloid-PET uptake.24. Potential intermediate mechanisms linking OSA and AD, such as decreased non rapid eye movement (NREM) slow wave activity (SWA) and increased inflammation affect amyloid and tau pathology,25,26 are associated with changes in cognition in late-life,27 and are more burdensome in blacks.28 OSA effects changes in circulating levels of CRP, TNFa, IL-6, and IL-17A.29,30 . More importantly, inflammation arising from cumulative stress exposure placed blacks at a greater risk for developing vascular risk factors,31-33 that increase AD-risk. In addition, SES and psychosocial factors11,34-36 may contribute to increase OSA and AD-risk in blacks. This highlights the need to utilize OSA as a unique disease model to explore racial differences in AD biomarkers.
Mechanisms of Sleep Deficiency and Effects on Brain injury and Neurocognitive Functions among Older Blacks
The multi-disciplinary team will utilize innovative dynamic and geospatial modeling in a multi-level framework to delineate the psycho social and environmental determinants of SD and its putative effects on the brain health of older blacks.
Mechanisms of Vascular Aneurysms
Our mission is to enable the use of human tissues with outcome data by basic and clinical investigators for research into the biology, causes, prevention and therapy of patients with documented arterial pathology (e.g. aortic dissection, lower extremity aneurysm, aortic aneurysm). Paired clinical information about disease history and research into vascular events will enable clinically relevant findings. We will prospectively collect both tissue samples and blood samples to study the mechanisms of the natural history of aneurysms and potential circulating biomarkers. We will be collaborating with Dr. Gelb in order to obtain tissue and blood from healthy controls during the organ procurement. We will also collect relevant clinical and medical information on these subjects and continue to collect follow-up information on the surgical patients using their medical records in order to follow their outcome. The tissue and blood will be stored indefinitely.
Mechanistic basis for impaired B cell depletion after anti-CD20 treatment of African American with neuro-inflammatory disorders
Infusable anti-CD20 (aCD20) therapies (rituximab, ocrelizumab) emerged as the mainstay of treatment in multiple sclerosis and related disorders (MSARD). Standard, every-6-month dosing of aCD20 achieves complete blood B-cell depletion in 99% of patients. However, some patients do not have complete B-cell depletion, which we postulate results from genetic polymorphisms influencing antibody-dependent cell cytotoxicity, complement-dependent pathways, or other factors. To unravel the mechanistic basis for early B-cell repletion, we will perform in-depth microfluorometric analyses of blood samples, BAFF cytokine measurement, and also study genetic polymorphisms relevant to B-cell biology using comprehensive Illumina genetic chip. Identification of mechanism(s) responsible for accelerated repletion, which may reflect suboptimal treatment, may enhance our understanding of how B-cell depletion is achieved with aCD20 therapies, and lead to personalized treatment regimens.
Medicaid Reenrollment Services | NYU Langone Health
NYU Langone, in partnership with Public Health Solutions, provides in-person, telephone, and web-based assistance with Medicaid reenrollment.
Medical & Surgical Procedures for Rosacea | NYU Langone Health
If symptoms of rosacea are resistant to medications, NYU Langone dermatologists may recommend a procedure like laser therapy.